We
just have to close the process of the complete disease now by integrating
one of its essential components : the infectious phenomenon. It is the
object of the fourth law, Dr. Hamer formulated as : THE
ONTOGENETIC SYSTEM OF THE MICROBES.
The terms system and ontogenetic
refer to their usage in the third law. System : because it brings an extended synthesis of the role played
by the microbes in the diseases. Ontogenetic
: because the different microbes being the fungi, the bacteria and
the viruses all have what is called a “tropism”, i.e. an affinity for
the tissues derived from a same embryonic origin. We will not have enough
time to develop this more technical aspect of the affinities, but this
is not at all necessary to understand the essence, i.e. the real role
of the microbes. And, here again, the examination of the facts will
throw over all the dogmas related to their noxiousness, contagion, prevention,
vaccines, etc.
Contrarily to what is thought,
microbes are not enemies that have to be fought by a defence system
being our immune system. They are, on the contrary, “friends” being
there to help
us recover more completely but also more violently. Example : if
you have developed a conflict affecting the biliary ducts of the liver,
you will develop hepatitis being the recovery phase, when you will have
solved this conflict. If you have viruses, your hepatitis will be more
severe, but more complete. If you do not have viruses at your disposal,
you will nevertheless develop your hepatitis. One will simply find no
trace of viruses in the serology in the entire liver-repairing inflammation
process. The viruses are thus not responsible for the hepatitis, it
is our organism that uses them to optimise recovery!
This notion of microbial aid
rests on concrete and repetitive observations, observations I do not
at all ask you to believe – as for the biological laws – but to verify
by yourself. First observation : the
microbes only intervene when the conflict is solved and only during
the second phase, thus on a previously modified tissue. In other
words : you can not develop an infection if your conflict is not solved.
Be it a furuncle, an angina, a bronchitis, a zona or the severest form
of tuberculosis, there is no exception whatsoever. This contradicts
the dogma of contagion with a healthy person, a contagion that could
never be explained by universal and constant criteria : we understand
it now.
I often hear following reasoning
: “I was very tired since a couple of days or a week ; it is normal
that my immune system was weakened and that I got the infection of X,
or the first microbe that crossed my path”. This reasoning is the echo
– deeply rooted in our beliefs – of these dogmas on microbial noxiousness,
contagion and immunity conceived as sole defence. The manipulation of
the biological laws brings us a much more reliable and at all times
reproducible explanation. If that fatigue was not the banal consequence
of extra work – which has nothing conflictual and can not make anyone
ill – it was part of the non-specific second phase symptoms, where our
nervous system commands us a need for additional rest. Simultaneously,
the reparation mechanisms set into action. I already told you that this
second phase is more than often more uncomfortable than the first one,
and, in case of microbial work, it will always be that way. Next, the
organ affected with infection will correspond to the type of conflict
that is solved. The close analysis of each case of infection replaces
those common beliefs on small and important depressions of our immune
system, as well as the classical chills, “responsibles” as uncertain
as non-repetitive.
If the microbes only intervene
in the reparation phase, one may already think they participate in the
restoration of the organs. But this does not yet formally constitutes
a proof, since one may interpose as an objection that they remain a
nuisance appearing only in the second phase, the objection being ill-assorted
though, because why only in the second phase? It is the second observation
that will establish their usefulness in the recovery phase : examine
what they really do. The observation is then very expressive. One sees
them destroying the proliferations having developed in the tissues reacting
by means of this kind of modification in the conflictual phase.
After the solution of the conflict, those proliferations have no further
object, and if antibiotics or equivalents do of course not fight the
microbes, they will try to restore the organ in the state preceding
the conflict. If the microbes are missing, or have been artificially
eliminated, those proliferations will stop but they will remain encysted.
The destruction may not be completed, but sufficient to enable a better
functioning of the organ. For example, an intestinal tumour may not
be entirely suppressed, but sufficiently necrosed to set aside all risk
of occlusion. Sometimes, their destructive action will leave a hole
in the organ, as it is the case in the tuberculous caverns. But, keeping
strictly to the facts, without “judging” their action, is it not better
to remain with an anodyne small tissular shortage rather than with a
tumour that could provoke compression phenomena? And, let us not forget
that, even the classical lung specialists, considered their patient
recovered when he only presented that sole cavern without any other
symptom.
On
the contrary, in tissues reacting by a loss of substance during the
conflictual phase, the microbes contribute to a destruction limited
to the cells which are no longer viable, to contribute afterwards to
the reconstruction concretely consisting here in a cellular proliferation. It is the reason why one
can only find for example a virus in the liver in case of hepatitis
when it is in its second phase, or in the so-called uterine cervix cancer evenso in reparation, after the ulceration of the conflictual
phase.
Let us finally add that in
the two types of tissular modification where the microbes intervene
in the reparation phase, their “work” does not systematically last during
the entire second phase, but only in function of their own action :
destruction, clearing up, reconstruction, etc. Let us take the cases
of pulmonary tuberculosis and purulent otitis : the secretions witnessing
the destruction stop before the end of the complete disease. The reason
is that all the reparation is performed in a liquid environment – an
environment constituting the two thirds of our organism where life develops!
– and that the complete restoration of an organ must go through the
elimination of this temporary liquid excess to come to the final cicatrisation
step. At the end of the second phase, the action of the microbes is
less necessary and even useless.
I, once again, resumed this
fourth law as there would be much more to precise, particularly the
actions more specific to each kind of micro-organism. But, considering
these simple and constant criteria, verifiable by their presence, may
we be satisfied with concepts as gratuitous as noxiousness in se, a
completely hazardous contagion or a prevention that is not confirmed?
On the contrary, the microbial actions is part of
the tremendous natural programmation of the disease and it is the
central computer, being our brain, that will decide of their appearance
as soon as the conflict is solved, as well as of their disappearance
as soon as their mission is accomplished.
To understand the importance
of an infection, you always have to do the same step : know the importance
of the conflict. And there will be nothing mysterious anymore in the
considerable variations that the same infection may take from one individual
to the other. Pulmonary tuberculosis, for example, may be a simple fortuitous
discovery on an X-ray in industrial medicine, or during a check-up.
You are being told : “Well, you have a tuberculous primary infection!”
You are rather astonished but the proof is there : one can still see
some typical small micro-calcifications. The radiologist reassures you
by speaking of an old story without any importance, and, if you are
curious, you will search in your past. It maybe happened when you were
17 or 18, a time where your missed school during a month, where you
have coughed and expectorated very much, had fever. But the parents
did not really worry, as everything was all right very quickly. And
that same tuberculosis can make you think of certain Italian operas,
where one spits out one’s lungs during a whole year, before dying of
exhaustion in the arms or one’s beloved, the vocal cords still in top
condition for the necessity of the cause… I try to ironise somewhat
her in order to make it less dramatic, but if the tuberculosis appears
to be lethal by its extent, you will always find a conflict in proportion.
Think about the outbreaks of tuberculosis during both world wars : it
was not a matter of malnutrition but rather an evident rise of the conflicts
of fear to die. The underfeeding is especially a limiting factor meant
to assume physically whatever important infection.
I leave the field of the infections
for a while to draw a parallel with an entirely different pathology,
because that one too can be banal or lethal, and because its explanation
invariably goes through the biological laws. Why can an infarction pass
unnoticed and be discovered accidentally during a cardiologic examination,
or kill the same day? Always the same answer : look for the conflict.
In the first case, it will only have lasted a few days or weeks and,
in the other case, at least eight or nine months. Let me add a precision
: the infarction is not due to the cardiac lesion, but to the reparation
oedema in the brain. This oedema compresses the area managing the cardiac
rhythm and its functioning, and, at its maximal extension, provokes
a blocking of the heart. It is one of the second phase cerebral complications
Dr. Hamer called the epileptoid crisis. Experimentation was even performed
on animals where several coronary arteries were brutally clamped and
the heart kept on beating. Personally, I have often seen patients, having
more than half of their coronary arteries obstructed, living a normal
life. A substitution circulation is foreseen everywhere. The infarction
is explained by the conflict and not by the arteriosclerosis and the
obstruction of the arteries.
Let
us come back to the infectious phenomenon in order to divest the so
usual notions of relapse and ill-treated infections
of their mythical quality. The term relapse may cover two scenarios.
On the one hand, it
effectively concerns a relapse in its strict sense, but in this case, after a simultaneous
relapse of the first conflictual phase. One may thus have, more or less
frequently, angina, bronchitis or vaginitis. Using the four laws, one
will examine why the person always falls back into his conflict, and
one will help him find the better parade for him. Result : done with
the infections, the consultations, the drugs and … the growing anxiety
over an innocent immune system heading directly to the charge of deficiency!
On the other hand,
it concerns a new attack of infection each time the anti-infectious
treatment is stopped. This time, it does not concern a relapse,
but the continuation of the microbial work having been contradicted,
and even completely masked, as long as the second phase was not finished.
One may compare this to an eczema becoming red and itchy again, when
one decides to stop the cortisone ointment … or to a leukaemia relapse,
when the bone marrow tries to regenerate after the last bludgeoning
of chemotherapy! The problem is that the doctor does not know how long
he will have to treat, since he does not know the duration of the first
phase. And all the doctors are confronted with the same problem, questioning
themselves on their greatest successes or failures ; especially when
the second phase is extremely long. If they knew its duration and if
they only wanted to succeed, they only would have to take in charge
those patients being very near to the end of their reparation phase,
because it is the last therapeutist consulted in the second phase who
wins the therapeutical praise! This happens of course in the great majority
of cases, since it is the reparation that is most often uncomfortable.
I have practices medicine during fifteen years without understanding
anything and I learned more in six months than in fifteen years, by
being able to “dismantle” the disease and thus to better foresee it
and to know where the patient is going to. In a few words, if you want
to know why your cystitis necessitated a package of antiseptics, a tube
of homeopathic granules, an acupuncture session, an osteopathic manipulation,
etc. or if one of those techniques had to be renewed 2, 5 or 8 times,
consider exactly the duration of your conflict.
As far as ill-treated
infections are concerned, it are conflicts
having come to a balance where each period of relief will see the microbial
work increase or start again. Here is an example that will show you
simultaneously the “shortcuts” one may use when questioning a patient.
When I see a man affected with chronic bronchitis since ten years, because
ill-treated at the time of course, I am not going to start by asking
him what he experienced ten years ago and that is still going on today.
Except, of course, if he is really used to the approach. I am first
going to ask him if he often needs sick-leaves and coughs less during
weekends and holidays ; or if it is rather the contrary, having his
weekends wasted by a worsening of the symptoms. If the answer is clear
enough, I already know where I have to go on searching for the conflict
: in the first eventuality, it will be the family environment and, in
the second one, the professional environment. And, I will add in the
same optic that, to evaluate if a retirement is a catastrophe or a blessing,
one should not be satisfied with the sole consideration of the job the
patient will no longer have…
I
will end this last law with a precision, essential for its correct application
: the eventual – and sometimes vital – necessity to interfere
in the infectious phenomenon. We have already
seen that an intervention in the natural phenomena may be mandatory,
such as the operation of a tumour endangering a patient’s life ; and
that may be the case in each of both phases. In the tumoral phenomenon,
this necessity is relatively seldom and most often justifies itself
by mechanical complications, such as obstruction or compression. In
the infectious phases, on the other hand, things are a little more complicated
even if the interventions must always obey imperatives of good sense
and – as we will see – perfectly comprehensible.
The first indication
is the risk for the patient of not being able to endure a “microbial
reparation work” too consistent for his physical possibilities. In the whole of these reparations, real
danger is seldom, but they have to be signalled. They will especially occur at both extremities of life, or when the
weakening due to microbial cleaning lasts longer consequently to a first
conflictual phase, too long itself ; and, a fortiori when both these
conditions are combined. Also when the individual is already weakened
for an entirely different reason than his infection: other disease,
malnutrition, etc. A few concrete examples : if the pneumonia after the solution of
a conflict of fear to die can not be circumvented, a baby or an elderly
will be less able to assume the continuous efforts of an expectoration,
which could lead, to choking. Evenso, one can not let a diarrhoea evolve
that would lead the diseased to a fatal state of dehydration.
The second indication
is intrusion, always in the second phase, of microbes not foreseen in
the programme. At this stage,
some explanation is necessary. First, in the immense majority of cases,
when being in an infectious phase, we use our own microbes. So, our
skin is covered with staphylococcus and our throat with streptococcus,
and when we develop a furuncle or an angina, what do we find? Staphylococcus
and streptococcus respectively! In a much larger number and considered
pathogenic. In reality, there are no good microbes becoming bad, but
simply an important multiplication only at the site where they have
to perform the reparation. Another example : in the urine of a woman
developing cystitis, colon bacillus is mostly detected, a bacterium
possessed by each one of us. But its concentration will exceed the limit
of 100,000 units, making it the responsible enemy to combat. While if
less than 10,000 units are found, be there infection or not, it will
be declared innocent. The incomprehension of the infectious phenomenon
engendered the creation of these arbitrary levels, because why not 80,000
or 50,000, values which are also observed? All this reminds us of other
more dramatic criteria, such as the one already exposed, being the simple
classification between non-malignant and malignant cells. The same lacking
engenders all these frozen classifications : the absence of observation
of the large variations in biological modifications and the evenso extended
one’s in the conflicts.
Next
to the microbes inherent to our organism and always ready to intervene,
others are existing reigning in the endemic state, i.e. usual to our
regions, but that will only “invade” us in case of more sporadic or
more important reparations. This is the case of numerous viruses and
several bacteria among which one deserves to be cited considering its
performances : it is the famous tubercle bacillus, the “Tuberculous
Koch Bacillus”. Fortunately gifted with an excellent “resistance”, this
ancient bacterium is especially appropriated to destroy the proliferations
developed in the tissues depending on the archaic brain area. It thus
contributes to clean the alveolar, intestinal, hepatic, genital, etc.
tumours. But the evenso performing “price put on its head” has largely
lessened its resistance and it has less and less the possibility to
help us. It is on purpose that I cited this micro-organism considered
a plague, though perfectly adapted to our occidental civilisation :
the example will serve to introduce the second justification to consider
intervention in the infectious phase.
This
indication derives from taking into consideration a
microbial ecosystem distributed over the world. It means that the numerous
microbes are spread according to the sometimes totally different climatic
zones. As long as an individual develops his
infection with a microbe belonging to his usual environment, a problem
may only occur in the situation cited higher of a reparation being too
difficult for him ; because of his age, his weakness or because of the
extent of his preceding conflictual phase. But the appearance of the
rapid moves to far away regions makes us come into contact with another
ecosystem the microbes of which are not adapted to the biological programme
having developed in our original environment. So, the germs to which
the natives of Central Africa or of the tropical American areas are
used are not at all adapted to the occidental Europeans and this is
where the system may fail. And vice versa : when the measles epidemics
spread with the American Indians, mortality proved to be very important
among the adults as the measles virus was not foreseen for these populations
; and only with the adults as the measles are a solution phase programmed
for childhood where it is harmless.
